RNA-Based Vaccine Technology: The Trojan Horse Did Not Contain mRNA

It Contains modRNA That Genetically Manipulates Healthy Cells

"Even though modRNA encodes for the spike protein of SARS-CoV-2, the aim was never to mimic viral RNA, which our immune system would immediately destroy, but to adapt it to the structure of human mRNA."

https://www.theepochtimes.com/health/rna-based-vaccine-technology-the-trojan-horse-did-not-contain-mrna_5195804.html

Also see:
https://t.me/robinmg/29871

Also read:
https://tomrenz.substack.com/p/mrna-and-why-it-matters

MRNA MODRNA & Why It Matters

"Remember, all the COVID vaccines are gene therapy products and none are using natural mRNA. Instead each of these are [modRNA] toying with those 3300 billion lines of code that make humans work and we are just hoping that there are no unintended consequences."

https://tomrenz.substack.com/p/mrna-and-why-it-matters

Moderna's mRNA Mystery

Some things don't add up

"Moderna, the producers of the mRNA-1273/Spikevax vaccine, are the ones principally responsible for the development of the mRNA transfection tech currently used in various COVID-19 vaccines.

The seed of the idea that would germinate into Moderna was planted in 2005, when Derrick Rossi read a paper written by Hungarian scientist Katalin Karikó on how nucleoside-modified mRNA could be made to evade human immune responses from toll-like receptors

The paper in question was published in Cell, back in 2005:

Suppression of RNA Recognition by Toll-like Receptors

According to this paper, using pseudouridine in place of uridine allows foreign mRNA to escape detection by TLR7/8

Toll-like receptors are a type of PRR, or pattern-recognition receptor, the smoke alarms of human cells. Their purpose is to detect molecular signs of damage or foreign objects (DAMPs and PAMPs) and induce an inflammatory response…"

👉 Read more: Spartacus

Suppression of RNA Recognition by Toll-like Receptors: The Impact of Nucleoside Modification and the Evolutionary Origin of RNA

"DNA and RNA stimulate the mammalian innate immune system through activation of Toll-like receptors (TLRs). DNA containing methylated CpG motifs, however, is not stimulatory. Selected nucleosides in naturally occurring RNA are also methylated or otherwise modified, but the immunomodulatory effects of these alterations remain untested.

We show that RNA signals through human TLR3, TLR7, and TLR8, but incorporation of modified nucleosides m5C, m6A, m5U, s2U, or pseudouridine ablates activity.

Dendritic cells (DCs) exposed to such modified RNA express significantly less cytokines and activation markers than those treated with unmodified RNA.

DCs and TLR-expressing cells are potently activated by bacterial and mitochondrial RNA, but not by mammalian total RNA, which is abundant in modified nucleosides.

We conclude that nucleoside modifications suppress the potential of RNA to activate DCs. The innate immune system may therefore detect RNA lacking nucleoside modification as a means of selectively responding to bacteria or necrotic tissue."

https://www.cell.com/immunity/fulltext/S1074-7613(05)00211-6

A "turbo-cancer" explanation:

Suppression of RNA Recognition by Toll-like Receptors

According to this paper, using pseudouridine in place of uridine allows foreign mRNA to escape detection by TLR7/8

Toll-like receptors are a type of PRR, or pattern-recognition receptor, the smoke alarms of human cells. Their purpose is to detect molecular signs of damage or foreign objects (DAMPs and PAMPs) and induce an inflammatory response…"

Paper extract here:

https://t.me/robinmg/29881

Syptoms of (early) SARSCoV2 not dissimilar to HCoV OC43

Along with HCoV-229E, -NL63 and -HKU1, HCoV-OC43 is responsible for up to 30% of all common colds in adults; however, more severe forms of these viral infections can lead to upper respiratory tract disease.

"The similarity in clinical symptoms of patients with both HCoV-OC43 and HCoV-229E often cause these infections to be indistinguishable from each other; however, sore throat manifestations are more commonly associated with HCoV-OC43 than HCoV-229E, which typically causes a greater extent of nasal inflammation.

Although most of the replication of HCoV-OC43 occurs in the epithelial cells of the upper respiratory tract, this virus is unique in its ability to tolerate mutations and even cause infection in the brain.

Rather than utilizing the BAX protein to induce apoptosis within neuronal cells, research has shown that HCoV-OC43 infection within the brain is most likely due to the necroptosis-inducing actions of the RIP1 and MLK1 proteins.

The ability of HCoV-OC43 to enter and infect neurons has been shown to induce acute encephalitis in mice that can be accompanied by various neurological disabilities post-infection, some of which include abnormal limb clasping reflex and decreased motor activity.  

In addition to its most notable effects on the upper respiratory tract and possible neuronal infiltration, a 2001 outbreak of HCoV-OC43 in Normandy, France found that this virus can also cause a wide range of digestive problems to occur.

More specifically, this outbreak found that up to 57% of patients with HCoV-OC43 experienced vomiting, diarrhea, and abdominal pain.

In addition to upper respiratory tract effects, patients with HCoV-OC43 in this French study also experienced several lower respiratory tract symptoms, of which included bronchitis, bronchiolitis, and pneumonia in 17%, 1-%, and 6.7% of all patients."

https://www.news-medical.net/health/What-is-OC43.aspx

For (early: 2019 or January 2020) Covid19 I would add to the descriptions in the link severe fatigue with muscle ache (unbearable) and joint ache, up to finger joints, and VERY high temperature (unprecedented).

The rest is VERY similar...

HCoV OC43 symptoms are very similar to early SARSCoV2:
https://t.me/robinmg/29883

How does HCoV-OC43 enter cells?

"The entry of HCoV-OC43 into human cells is largely achieved through the caveolin-1-dependent pathway of endocytosis; however, virus-containing vesicles at the cell surface can also undergo scission to also penetrate human cells.

Notably, while host factors like interferon-inducible transmembrane proteins (IFITMs) often prevent the entry of coronaviruses like HCoV-229E, -NL63, SARS-CoV and MERS-CoV from entering cells through its various antiviral functions, IFITM2 and IFITM3 promote the entry and subsequent infection of HCoV-OC43 into human cells."

https://www.news-medical.net/health/What-is-OC43.aspx

I have said this since at least 2021.

This is where many in the freedom movement got confused:

1. Novel does not mean more lethal, a novel virus can be absolutely harmless.

2. There was no difference in lethality between SARSCoV2 and common circulating betacoronaviruses like OC43.

Therefore mortality data has always been irrelevant. Too many fell in this trap.

The question still remains, how did so many people get to have their most symptomatic respiratory infection in 2019, 2020 & 2021?

REFERENCE
“We have characterized an outbreak of respiratory illness due to HCoV-OC43. The observed attack rate of 67% and case fatality rate of 8% underscore the pathogenic potential of HCoVs in frail populations. This adds to other observations underscoring that CoVs other than SARS-CoV may be responsible for a broader spectrum of disease”
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2095096/

Amended letter to decline child flu vaccine with working footnotes 3 and 4

@lawyersoflight

https://x.com/0vernite/status/1696588956328964321?s=46&t=WSMLCQNckomGtNidcKiTmw

Please see tweets in reply to the news conference in Japan.

Please spread this short video because mainstream media won’t.

YOU ARE THE VIRUSES THEY WANT TO REDUCE

ZERO VIRUSES = ZERO HUMANS

The demonization of viruses follows the same logic as the demonization of CO2. Viruses are symbiotic and essential to human life, and plant life too. They are not by definition harmful, far from it. Yet again the false demonization only benefits the depopulation agenda.

Move Over, Bacteria! Viruses Make Their Mark as Mutualistic Microbial Symbionts | Journal of Virology
"Viruses are being redefined as more than just pathogens. They are also critical symbiotic partners in the health of their hosts. In some cases, viruses have fused with their hosts in symbiogenetic relationships. Mutualistic interactions are found in plant, insect, and mammalian viruses, as well as with eukaryotic and prokaryotic microbes, and some interactions involve multiple players of the holobiont.

Symbiosis and symbiogenesis.
The mammalian genes for syncytin, essential in the establishment of the placenta, are retroviral env genes of viruses endogenized on several different occasions (10) and even function differently in ruminants versus other mammals (11). Many other symbiogenic viruses are integrated into the host genome. The role of these viruses, including many viruses beyond the retro- and pararetroviruses, is occasionally known. The endogenization events are often ancient, and these elements are considered viral fossils that can help us understand the deep evolution of viruses.

Good viruses in mammalian health.
The guts of humans and other mammals are rich in viruses. These are largely still in the discovery phase. Many of the gut viruses are bacteriophage, although eukaryotic viruses are also part of the human virome, and these include both human viruses and viruses of eukaryotic symbionts (23). For the most part, little is known about how these viruses impact their hosts, but their ubiquitous presence implies functions. In some cases, phage may be regulating the populations of resident bacteria or they could affect the expression of bacterial genes that are involved in host digestion (24). In a recent study, bacteriophage were shown to adhere to mucous membranes in many different metazoan hosts. Mucous membranes are found at the point of entry of many bacterial pathogens. The phage are poised to provide the first line of defense against bacteria invading the metazoan host by infection and lysis. The metazoan host is benefitted by this early attack of potential pathogens, and viruses are benefitted by access to new hosts

Conclusions
Viewing viruses in the context of ecology (28) provides a framework for a deeper understanding of the intertwined relationships of all life, including viruses. It is inevitable that more examples of mutualistic viruses will be revealed as we continue this exciting phase in virology of virus discovery."

https://journals.asm.org/doi/10.1128/jvi.02974-14

A contact on a Telegram channel recently made me aware that a certain allegedly terrorist event in London in July ‘05 was nothing like as portrayed by our lovely media. 

I wonder if you already knew about this. Probably not. I’d not previously heard it alleged that it was an inside job.
There’s no doubt about it, it was. See what you make of the evidence.

This home made one hour documentary was so much of a threat to the perpetrators of the outrages committed on that day that, once copies of it were sent to the judge, jury and others involved in a related criminal trial, sent with the intention of heading off a miscarriage of justice, the author of the documentary was arrested.

Five months later, he was tried on trumped-up charges including, with dark humour, that his actions “might undermine public confidence in the government and security apparatus in U.K.”

I also attach a link to the story of that trial (25min only). It turns out that, having seen this documentary, the jury refused to convict the defendant (the film’s author), even when instructed by the judge to bring a majority, rather than a unanimous, verdict.
This tremendously embarrassing verdict tacitly tells us that the evidence that the events were an inside job is overwhelming. 

I am no longer surprised to learn this. I remember being deeply sceptical about the story we were told by media types, government and various spokespersons. I reasoned at the time that, if the narrative was as told to us, a large number of similar events were to be expected, since the effort involved was at the lower end of cost and complexity. I then walked backwards in time, arguing that, if it was as simple to do as we were told, similar attacks it would already have happened. They hadn’t happened & subsequently did not happen, either. 

Incidentally, I was given a link to a YouTube video. Of course, that’s been deleted. However, using minor search engines, I was easily able to find this documentary, but only because I knew what to look for.
The documentary:

https://archive.org/details/7-7_Ripple_Effect

The initial message containing the story of the trial of the author of the documentary:

https://inoneplace.com/death-taxes-a-new-short-film-by-tony-rooke/

My wife, watching these videos with me, observed the close similarities to the covid situation. As with covid, the diabolical minds who arranged one of the worst alleged acts of terrorism in U.K. history sought to tell us what they were going to do. They held a tabletop simulation of the outrage a year before the event and even televised it under the documentary banner, Panorama. Consider Event 201 in relation to covid a few months later. Same M.O. 
(As an aside, I remind people that Event 201 was claimed to be a fictional emergence, in China, of a novel, severe, acute respiratory virus, the type being a coronavirus. Even now, you’d have to be extraordinarily tolerant of unlikely coincidences not to recognise, at least in retrospect, that the entire event was “an inside job”).

Worse still, on the day of the actual outrage, the authorities claimed to have been running a response rehearsal of an almost identical, multiple-location terrorist attack on London. What are the chances? Finally, at this time, we have probably the best explanation for the shooting dead of a Brazilian contract electrician shortly thereafter. The perpetrators lack imagination. This practise of recruiting participants at some level & then murdering them is an old technique that we have seen before.

With best wishes,
Mike 

🎞 ​​With LESS THAN A WEEK to go before the River of Freedom FIlm premieres, here's our 60" trailer!

https://youtu.be/jGT2TdjWz3o

RIVER OF FREEDOM 🇳🇿
60" FILM TRAILER
@River_of_Freedom
@RobinMG
https://x.com/robinmonotti/status/1696763633101930692

Viruses are essential to human life. Their demonization together with their denial are an oligarchic psyop, the real threat to biological knowledge.

Read more:
https://t.me/robinmg/29890

https://twitter.com/robinmonotti/status/1696789027989389449